An important role here is played by the level of beta-amyloid 42 in human cerebrospinal fluid.
Scientists say that the number of plaques in the brain or the amount of residual beta-amyloid 42 plays a role in the development of Alzheimer’s disease. Science Alert writes about it.
Researchers analyzed data on a group of people with a rare inherited gene mutation that puts them at high risk of developing Alzheimer’s disease. In one experiment, depletion of amyloid beta 42 (the functional version of amyloid beta) was found to be more damaging than plaque counts (insoluble accumulations of amyloid beta).
The study took place over three years. Thus, scientists found that people with high levels of beta-amyloid 42 in their cerebrospinal fluid (fluid around the brain and spinal cord) were protected and their cognition was preserved during the study.
It is also noted that some drugs have been developed to lower levels of beta-amyloid 42, with the assumption that by lowering normal levels of the protein, patients will have less plaque buildup. Unfortunately, these drugs often worsen the patient’s condition.
Remember that last year, a group of American and Indian scientists discovered that the natural compound fenhol in basil leaves can protect the brain from Alzheimer’s disease.
Scientists have created a drug that slows the rate of memory loss
News from Correspondent.net on Telegram. Subscribe to our channel Athletistic
Source: korrespondent
